EVALUATION OF SURVIVAL AND NEURODEVELOPMENT IN NEONATES BORN VERY PRETERM AT A TERTIARY CENTRE IN PORTUGAL



Activation of exchange protein activated by cAMP in the rat basolateral amygdala impairs reconsolidation of a memory associated with self-administered copyright.

The intracellular mechanisms underlying memory reconsolidation critically involve cAMP signaling.These events were originally attributed to PKA activation by cAMP, but the identification of Exchange Protein Activated by cAMP (Epac), as a distinct mediator of cAMP signaling, suggests that cAMP-regulated processes that subserve memory reconsolidation

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